Author

A. Alase

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Immunology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:flow cytometry· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

A. Antanaviciute· 1 shared A. Psarras· 1 shared E. M. Vital· 1 shared G. C. Tsokos· 1 shared I. M. Carr· 1 shared M. Wittmann· 1 shared M. Y. Md Yusof· 1 shared P. Emery· 1 shared

Activity by Year

2018-2018 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 28 mutual co-author links

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1 shared paperImmunology

A. Antanaviciute

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1 paper

Recent·Most cited

Plasmacytoid dendritic cells are functionally exhausted while non-haematopoietic sources of type I interferon dominate human autoimmunity

A. Psarras, A. Alase, A. Antanaviciute, I. M. Carr, M. Y. Md Yusof, M. Wittmann, P. Emery, G. C. Tsokos, E. M. Vital

The paper shows that plasmacytoid dendritic cells are functionally exhausted in autoimmunity, and non-haematopoietic sources like keratinocytes drive type I interferon production.

bioRxiv · Dec 20, 2018Immunology