Author

A. Anginot

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Bonaud· 1 shared A. Pettit· 1 shared B. Jose· 1 shared B. Nowlan· 1 shared B. Schell· 1 shared C. Freitas· 1 shared C. Lobry· 1 shared C. Moulin· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

A. Bonaud

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

CXCR4 signaling strength regulates hematopoietic multipotent progenitor fate through extrinsic and intrinsic mechanisms

V. Rondeau, M. Kalogeraki, L. Roland, Z. A. Nader, V. Gourhand, A. Bonaud, J. Lemos, M. Khamyath, C. Moulin, B. Schell, M. Delord, G. Bidaut, S. Lecourt, C. Freitas, A. Anginot, , K. Balabanian

This study shows that CXCR4 signaling strength influences hematopoietic multipotent progenitor fate through both extrinsic and intrinsic mechanisms, with implications for myeloid skewing in WHIM Syndrome.

bioRxiv · May 31, 2023 · PreprintCell Biology

Spinal cord injury reprograms muscle fibro-adipogenic progenitors to formheterotopic bones within muscles

H.-W. Tseng, D. Girard, K. Alexander, S. Millard, F. Torossian, A. Anginot, W. Fleming, J. Gueguen, M.-E. Goriot, D. Clay, B. Jose, B. Nowlan, A. Pettit, M. Salga, F. Genet, , J.-P. Levesque

This study identifies fibro-adipogenic progenitors (FAPs) as the cells-of-origin for neurogenic heterotopic ossifications (NHO) following spinal cord injury, showing their reprogramming into osteoblasts.

bioRxiv · Nov 4, 2021 · PreprintPathology