Author

A. E. Ramer-Tait

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Evolutionary Biology· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1

Frequent Co-Authors

R. J. Schmaltz· 3 shared A. K. Benson· 2 shared H. Kittana· 2 shared J. Clarke· 2 shared J. M. Hostetter· 2 shared J. Walter· 2 shared R. R. Segura Munoz· 2 shared S. Mantz· 2 shared

Activity by Year

2017-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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3 shared papersEvolutionary Biology

R. J. Schmaltz

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Author profile →

3 papers

Recent·Most cited

Local adaptation of host-species specific gut microbiota

D. D. Sprockett, J. D. Price, A. F. Juritsch, R. J. Schmaltz, M. V. F. Real, S. Goldman, M. J. Sheehan, A. E. Ramer-Tait, A. H. Moeller

This study shows that natural selection in house mice drives the assembly of gut microbiota from native and non-native sources, with adaptive advantages for native bacteria.

bioRxiv · Aug 22, 2022Evolutionary Biology

Establishing the phenotypic basis of adherent-invasive Escherichia coli (AIEC) pathogenicity in intestinal inflammation

H. Kittana, J. C. Gomes Neto, K. Heck, J. Sughroue, Y. Xian, S. Mantz, R. R. Segura Munoz, L. A. Cody, R. J. Schmaltz, C. L. Anderson, R. A. Moxley, J. M. Hostetter, S. C. Fernando, J. Clarke, S. D. Kachman, , A. E. Ramer-Tait

This study investigates AIEC pathogenicity in intestinal inflammation by linking in vitro phenotypes to in vivo disease severity in a murine model.

bioRxiv · Sep 18, 2019 · PreprintMicrobiology

A gut pathobiont synergizes with the microbiota to instigate inflammatory disease marked by immunoreactivity against other symbionts but not itself

J. C. Gomes-Neto, H. Kittana, S. Mantz, R. R. Segura Munoz, R. J. Schmaltz, L. B. Bindels, J. Clarke, J. M. Hostetter, A. K. Benson, J. Walter, A. E. Ramer-Tait

This study shows that a gut pathobiont, H. bilis, synergizes with the microbiota to cause inflammation, triggering a Th17 response against other microbes rather than itself.

bioRxiv · Aug 18, 2017Immunology