Author

A. M. Bogis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Approach:experimental assay· 1 Organism:mouse· 1

Frequent Co-Authors

A. J. Lee· 1 shared A. S. Yee· 1 shared C. Carland· 1 shared C. G. Herbosa· 1 shared F. H. Deleso-Frechette· 1 shared G. Triantafillou· 1 shared K. E. Paulson· 1 shared K. Li· 1 shared

Activity by Year

2022-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

A. J. Lee

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1 paper

Recent·Most cited

CHA1: A New Combinatorial Therapy That Reciprocally Regulates Wnt and JAK/STAT/Interferon Signaling to Re-program Breast Tumors and the Tumor-Resident Landscape.

M. K. Alamoudi, M. E. Chipman, F. H. Deleso-Frechette, A. M. Bogis, R. S. Alqurashi, K. Li, R. Zhang, M. Castañer, G. Triantafillou, C. G. Herbosa, C. Carland, A. J. Lee, K. S. Gillani, K. E. Paulson, A. S. Yee

CHA1, a combination of EGCG and decitabine, reprograms TNBC tumors and their microenvironment by inhibiting Wnt and activating JAK/STAT/IFN signaling, enhancing PD-L1 expression and T-cell infiltration.

bioRxiv · Mar 25, 2022 · PreprintCancer Biology