Author

A. M. Dylag

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Assay:metagenomics· 1

Frequent Co-Authors

G. S. Pryhuber· 2 shared A. Grier· 1 shared A. L. Gill· 1 shared A. McDavid· 1 shared A. R. Falsey· 1 shared D. J. Topham· 1 shared H. A. Kessler· 1 shared H. Huyck· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersPhysiology

G. S. Pryhuber

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2 papers

Recent·Most cited

Low Dose Hyperoxia Primes Airways for Fibrosis in Mice after Influenza A Infection

A. M. Dylag, J. Haak, R. Warren, M. Yee, G. S. Pryhuber, M. A. O'Reilly

The paper shows low-dose neonatal hyperoxia increases TSP-1 expression in mice, which primes lungs for fibrosis after influenza A infection, even when lung function appears normal.

bioRxiv · Jun 17, 2020Physiology

Aberrant newborn T cell and microbiota developmental trajectories predict respiratory compromise during infancy

A. Grier, N. Laniewski, A. L. Gill, H. A. Kessler, H. Huyck, J. Holden-Wiltse, S. Bandyopadhyay, J. Carnahan, A. M. Dylag, D. J. Topham, A. R. Falsey, M. T. Caserta, G. S. Pryhuber, S. R. Gill, A. McDavid, K. M. Scheible

The paper identifies that prenatal antibiotics or infection disrupt T cell and microbiota development, increasing respiratory morbidity risk in infants.

bioRxiv · Jun 16, 2020 · PreprintImmunology