Author

A. Pantazis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Developmental Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Assay:electrophysiology· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:long-read sequencing· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

A. W. Mould· 1 shared C. M. Sellgren· 1 shared Erik Smedler· 1 shared L. Jonsson· 1 shared M. Celik· 1 shared M. Landen· 1 shared M. Nikpour· 1 shared N. A. L. Hall· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperDevelopmental Biology

A. W. Mould

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Developmentally-regulated splice remodeling shapes functional CACNA1C channel diversity during human neurodevelopment

M. Celik, O. A. Arshad, N. A. L. Hall, S. S. Yasarbas, M. Nikpour, L. Jonsson, C. M. Sellgren, M. Landen, A. Pantazis, P. J. Harrison, A. W. Mould, Erik Smedler

This study identifies developmentally-regulated alternative splicing of CACNA1C during human neurodevelopment and shows it affects CaV1.2 channel function.

bioRxiv · Nov 9, 2026 · PreprintDevelopmental Biology