Smc5/6-antagonism by HBx is an evolutionary-conserved function of hepatitis B virus infection in mammals
F. Filleton, F. Abdul, L. Gerossier, A. Paturel, J. Hall, M. Strubin, L. Etienne
This study shows HBx from diverse hepadnaviruses inactivates Smc5/6 in mammals, suggesting this is an evolutionary-conserved antiviral mechanism.
bioRxiv · Dec 1, 2017Microbiology