Author

A. R. Mele

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

J. W. Adelsberger· 2 shared Q. Chen· 2 shared T. Imamichi· 2 shared B. Sowrirajan· 1 shared C. Watkins· 1 shared H. Sui· 1 shared J. Higgins· 1 shared J. Yang· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersImmunology

J. W. Adelsberger

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2 papers

Recent·Most cited

Interleukin-27-induced HIV-resistant dendritic cells suppress reveres transcription following virus entry in an SPTBN1, Autophagy, and YB-1 independent manner

T. Imamichi, Q. Chen, B. Sowrirajan, J. Yang, S. Laverdure, A. R. Mele, C. Watkins, J. W. Adelsberger, J. Higgins, H. Sui

This study shows that IL-27 induces HIV-resistant dendritic cells that suppress reverse transcription post-virus entry, independent of SPTBN1, autophagy, and YB-1.

bioRxiv · Jun 12, 2023Immunology

S100A8 and S100A9, biomarkers of SARS-Cov-2-infected patients, suppress HIV replication in primary macrophages

R. M. Oguariri, T. W. Brann, J. W. Adelsberger, Q. Chen, S. Goswami, A. R. Mele, T. Imamichi

This study shows that S100A8 and S100A9 subunits inhibit HIV replication in macrophages but not T cells, despite their role in SARS-CoV-2 infection.

bioRxiv · Oct 20, 2021 · PreprintMicrobiology