Author

A. Sanchez-Pozo

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1

Frequent Co-Authors

A. Gallardo· 2 shared A. Molina· 2 shared D. Landeira· 2 shared L. Lopez-Onieva· 2 shared A. Serrano-Prados· 1 shared E. Belmonte-Reche· 1 shared F. E. Cara· 1 shared H. G. Asenjo· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersCancer Biology

A. Gallardo

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2 papers

Recent·Most cited

EZH2 represses mesenchymal genes and upholds the epithelial state of breast carcinoma cells

A. Gallardo, L. Lopez-Onieva, E. Belmonte-Reche, I. Fernandez-Rengel, A. Serrano-Prados, A. Molina, A. Sanchez-Pozo, D. Landeira

The paper shows EZH2 represses mesenchymal genes and maintains the epithelial state in breast cancer cells during EMT, similar to its role in lung cancer.

bioRxiv · Mar 13, 2023Cancer Biology

EZH2 endorses cell plasticity to carcinoma cells facilitating mesenchymal to epithelial transition and tumour colonization

A. Gallardo, A. Molina, H. G. Asenjo, L. Lopez-Onieva, J. C. Alvarez-Perez, J. Martorell-Marugan, F. E. Cara, M. Espinosa-Martinez, P. Carmona-Saez, S. Granados, P. Medina, A. Sanchez-Pozo, D. Landeira

This study identifies EZH2 as a master regulator of EMT in carcinoma cells, showing it represses mesenchymal genes and facilitates tumour colonization.

bioRxiv · Mar 11, 2022 · PreprintCancer Biology