Author

A. Spengler

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:mouse· 2 Assay:scRNA-seq· 2 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1 Assay:flow cytometry· 1 Organism:human· 1

Frequent Co-Authors

J. Domenico· 2 shared L. Gapin· 2 shared L. Loh· 2 shared C. Levens· 1 shared D. Orlicky· 1 shared H. S. Krovi· 1 shared H. V. Meyer· 1 shared J. D. Torres· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersImmunology

J. Domenico

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2 papers

Recent·Most cited

Unraveling the Phenotypic States of Human innate-like T Cells: Comparative Insights with Conventional T Cells and Mouse Models

L. Loh, S. Carcy, H. S. Krovi, J. Domenico, A. Spengler, Y. Lin, J. D. Torres, W. Palmer, P. J. Norman, M. Stone, T. Brunetti, H. V. Meyer, L. Gapin

This study compares human innate-like T cells (Tinn) with conventional T cells and mouse models, revealing distinct transcriptional programs and developmental pathways.

bioRxiv · Dec 7, 2023Immunology

MAIT cells drive chronic inflammation in a genetically diverse murine model of spontaneous colitis.

L. Loh, D. Orlicky, A. Spengler, C. Levens, S. Celli, J. Domenico, J. Klarquist, J. Onyiah, J. Matsuda, K. A. Kuhn, L. Gapin

This study investigates the role of MAIT cells in a genetically diverse mouse model of spontaneous colitis, finding that MAIT cells drive chronic inflammation and colonic pathology.

bioRxiv · Nov 29, 2023 · PreprintImmunology