Author

A. Tendler

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Physiology· 2 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:statistical model· 3 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

U. Alon· 3 shared Y. Korem Kohanim· 3 shared A. Bar· 2 shared A. Mayo· 2 shared M. Raz· 2 shared O. karin· 2 shared T. Milo· 2 shared D. S. Glass· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 31 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersPhysiology

U. Alon

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Unifying design principles of endocrine gland mass and its regulatory circuits

M. Raz, T. Milo, Y. Korem Kohanim, O. karin, A. Tendler, A. Bar, D. S. Glass, A. Mayo, U. Alon

This study identifies unifying design principles of endocrine gland mass and regulatory circuits across human hormone systems using mathematical analysis.

bioRxiv · Jul 3, 2023 · PreprintPhysiology

A new model for the HPA axis explains dysregulation of stress hormones on the timescale of weeks

O. Karin, M. Raz, A. Tendler, A. Bar, Y. Korem Kohanim, T. Milo, U. Alon

This paper develops a new mathematical model of the HPA axis that explains dysregulation of stress hormones over weeks by incorporating gland mass dynamics and growth factor effects.

bioRxiv · Jan 1, 2020Physiology

Endocrine autoimmune disease as a fragility of immune-surveillance against hypersecreting mutants

Y. Korem Kohanim, A. Tendler, A. Mayo, N. Friedman, U. Alon

This paper proposes a theory that endocrine autoimmune diseases arise from a fragility in immune surveillance against hypersecreting mutant cells.

bioRxiv · Nov 17, 2019Immunology