Author

Abigail L. Hartz

Purdue University West Lafayette

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

Ashtyn N. Martens· 1 shared Hamood Rehman· 1 shared Hanan TlaisiD· 1 shared James F. MarkworthiD· 1 shared Vandré C. FigueiredoiD· 1 shared Xinyue LuiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

6 top collaborators · 15 mutual co-author links

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1 shared paperPhysiology

Ashtyn N. Martens

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1 paper

Recent·Most cited

C26 and CT26 colorectal cancer models exhibit divergent cachexia phenotypes, intramuscular inflammation, and protein turnover signaling

Xinyue Lu, Hanan Tlais, Hamood Rehman, Ashtyn N. Martens, Abigail L. Hartz, Vandré C. Figueiredo, James F. Markworth

This study compares C26 and CT26 colorectal cancer models, finding that C26 induces more severe cachexia, inflammation, and muscle protein turnover disruption than CT26.

bioRxiv · Apr 24, 2026 · PreprintPhysiology