Author

Aron Geurts

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1

Signature Assays, Organisms & Methods

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Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

David V. SerrezeiD· 1 shared Deanna J. Lamont· 1 shared Harold D. Chapman· 1 shared Ingo Schmitz· 1 shared Jennifer Allocco· 1 shared Jennifer R. Dwyer· 1 shared Jeremy J. Racine· 1 shared Jeremy J. Ratiu· 1 shared

Activity by Year

2018-2018 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperImmunology

David V. SerrezeORCID

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1 paper

Recent·Most cited

A hypermorphic Nfkbid allele represents an Idd7 locus gene contributing to impaired thymic deletion of autoreactive diabetogenic CD8+ T-cells in NOD mice.

Maximiliano Presa, Jeremy J. Racine, Jennifer R. Dwyer, Deanna J. Lamont, Jeremy J. Ratiu, Vishal Kumar Sarsani, Yi-Guang Chen, Aron Geurts, Ingo Schmitz, Timothy Stearns, Jennifer Allocco, Harold D. Chapman, David V. Serreze

This study identifies a hypermorphic Nfkbid allele at the Idd7 locus that impairs thymic deletion of autoreactive CD8+ T-cells in NOD mice, contributing to type 1 diabetes development.

bioRxiv · Mar 26, 2018 · PreprintImmunology