Author

B. Bea-Mascato

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Approach:graph neural network· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

D. Valverde· 2 shared A. Iglesias-Rodriguez· 1 shared C. Solarat· 1 shared E. Neira-Goyanes· 1 shared E. Petsalaki· 1 shared G. Giudice· 1 shared I. Pinheiro-de-Sousa· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 13 mutual co-author links

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2 shared papersCell Biology

D. Valverde

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Author profile →

2 papers

Recent·Most cited

Phosphoproteomic profiling highlights CDC42 and CDK2 as key players in the regulation of the TGF-β pathway in ALMS1 and BBS1 knockout models

B. Bea-Mascato, G. Giudice, I. Pinheiro-de-Sousa, C. Solarat, E. Petsalaki, D. Valverde

The paper identifies CDC42 and CDK2 as key regulators of the TGF-β pathway in ALMS1 and BBS1 knockout models through phosphoproteomic analysis.

bioRxiv · Nov 4, 2022 · PreprintCell Biology

Depletion of ALMS1 affects TGF-β signalling pathway and downstream processes such as cell migration and adhesion capacity.

B. Bea-Mascato, E. Neira-Goyanes, A. Iglesias-Rodriguez, D. Valverde

This study shows that ALMS1 depletion affects TGF-β signaling, leading to changes in cell migration, adhesion, and apoptosis in HeLa and BJ-5ta cells.

bioRxiv · Dec 19, 2021Cell Biology