Author

B. D. Standinski

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Assay:metagenomics· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

B. Miu· 1 shared E. S. Huseby· 1 shared J. Kang· 1 shared K. Sylvia· 1 shared M. Frascoli· 1 shared N. Malhotra· 1 shared N. Spidale· 1 shared

Activity by Year

2019-2019 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperImmunology

B. Miu

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Neonatal-derived IL-17 producing dermal gd T cells are required to prevent spontaneous atopic dermatitis

N. Spidale, N. Malhotra, K. Sylvia, M. Frascoli, B. Miu, B. D. Standinski, E. S. Huseby, J. Kang

This study shows that neonatal-derived IL-17-producing γδ T cells prevent spontaneous atopic dermatitis by maintaining skin homeostasis and preventing dysbiosis and inflammation.

bioRxiv · Jun 28, 2019Immunology