Author

B. Schuhmacher

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Organism:human· 1

Frequent Co-Authors

D. John· 2 shared G. Luxan· 2 shared S. Dimmeler· 2 shared W. T. Abplanalp· 2 shared A. Debes· 1 shared A. Fischer· 1 shared A. M. Zeiher· 1 shared A. Tamiato· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersPhysiology

D. John

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Author profile →

2 papers

Recent·Most cited

Age-dependent RGS5 loss in pericytes induces cardiac dysfunction and fibrosis in the heart

A. Tamiato, L. S. Tombor, A. Fischer, M. Muhly-Reinholz, L. R. Vanicek, B. N. Togru, J. Neitz, S. F. Glaser, M. Merten, D. Rodriguez Morales, J. Kwon, S. Klatt, B. Schuhmacher, S. Guenther, W. T. Abplanalp, , G. Luxan

This study shows that loss of RGS5 in pericytes with age leads to cardiac dysfunction and fibrosis by altering pericyte function and activating fibroblasts.

bioRxiv · Dec 8, 2023Physiology

DNMT3A clonal hematopoiesis-driver mutations induce cardiac fibrosis by paracrine activation of fibroblasts

M. Shumliakivska, G. Luxan, I. Hemmerling, W. T. Abplanalp, X. Li, M. Scheller, C. Muller-Tidow, F. Leuschner, B. Schuhmacher, A. Debes, S.-F. Glaser, M. Muhly Reinholz, K. Kirschbaum, J. Hoffmann, E. Nagel, , S. Dimmeler

This study shows that DNMT3A mutations in hematopoietic cells drive cardiac fibrosis by activating fibroblasts through paracrine signaling, contributing to heart failure progression.

bioRxiv · Jan 7, 2023Physiology