Author

C. C. Duwaerts

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Approach:experimental assay· 3 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Assay:flow cytometry· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. N. Mattis· 2 shared J. J. Maher· 2 shared A. Alkhani· 1 shared A. Nijagal· 1 shared C. Her· 1 shared C. Korsholm· 1 shared C. L. Her· 1 shared C. S. Levy· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersCell Biology

A. N. Mattis

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

ImmunologybioRxivPreprintJun 18, 2022

Neonatal hepatic myeloid progenitors expand and propagate liver inflammation in mice

A. Alkhani, C. Korsholm, S. Mohamedaly…·A. Nijagal

This study shows that neonatal liver inflammation in mice is driven by expanded hematopoietic progenitors, and their depletion prevents disease progression.

DOI 10.1101/2022.06.18.496674

mouseflow cytometry

Cell BiologyCell Death & DifferentiationPeer-reviewedNov 20, 2020

Hepatocyte-specific deletion of XBP1 sensitizes mice to liver injury through hyperactivation of IRE1α

C. C. Duwaerts, K. Siao, R. K. Soon…·J. J. Maher

This study shows that deleting XBP1 in hepatocytes causes liver injury in mice, which is due to excessive IRE1 activation rather than XBP1 loss itself.

DOI 10.1038/s41418-020-00671-1

mousebulk RNA-seq

PathologybioRxivApr 20, 2020

iPSC-derived hepatocytes from patients with nonalcoholic fatty liver disease display a disease-specific gene expression profile

C. C. Duwaerts, C. L. Her, N. J. Phillips…·J. J. Maher

The paper generates iPSC-derived hepatocytes from NAFLD patients to identify disease-specific gene expression profiles without metabolic challenge.

DOI 10.1053/j.gastro.2021.02.050

humanscRNA-seq