Author

J. J. Maher

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

A. N. Mattis· 2 shared C. C. Duwaerts· 2 shared C. Her· 1 shared C. L. Her· 1 shared H. Willenbring· 1 shared K. Kohno· 1 shared K. Siao· 1 shared N. J. Phillips· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersCell Biology

A. N. Mattis

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Author profile →

2 papers

Recent·Most cited

Hepatocyte-specific deletion of XBP1 sensitizes mice to liver injury through hyperactivation of IRE1α

C. C. Duwaerts, K. Siao, R. K. Soon, C. Her, T. Iwawaki, K. Kohno, A. N. Mattis, J. J. Maher

This study shows that deleting XBP1 in hepatocytes causes liver injury in mice, which is due to excessive IRE1 activation rather than XBP1 loss itself.

Cell Death & Differentiation · Nov 20, 2020Peer-reviewedCell Biology

iPSC-derived hepatocytes from patients with nonalcoholic fatty liver disease display a disease-specific gene expression profile

C. C. Duwaerts, C. L. Her, N. J. Phillips, H. Willenbring, A. N. Mattis, J. J. Maher

The paper generates iPSC-derived hepatocytes from NAFLD patients to identify disease-specific gene expression profiles without metabolic challenge.

bioRxiv · Apr 20, 2020Pathology