Author

C. E. Billiot

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 4

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

M. S. McDaniel· 3 shared N. Lindgren· 3 shared W. E. Swords· 3 shared N. A. Sumpter· 2 shared A. J. Barron· 1 shared I. F. Chaple· 1 shared K. N. Valladeres· 1 shared M. J. Gray· 1 shared

Activity by Year

2020-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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3 shared papersMicrobiology

M. S. McDaniel

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Comparative genomics of clinical Stenotrophomonas maltophilia isolates reveals regions of diversity which correlate with colonization and persistence in vivo

M. S. McDaniel, N. A. Sumpter, N. Lindgren, C. E. Billiot, W. E. Swords

This study compares the genomes of clinical S. maltophilia isolates to identify genomic regions linked to colonization and persistence in vivo, particularly in coinfection with P. aeruginosa.

bioRxiv · Jul 14, 2023 · PreprintMicrobiology

Pathogenesis of Achromobacter xylosoxidans respiratory infections: colonization and persistence of airway epithelia and differential gene expression in synthetic cystic fibrosis sputum medium

C. E. Billiot, M. S. McDaniel, N. Lindgren, W. E. Swords

bioRxiv · Apr 4, 2023 · PreprintMicrobiology

Pseudomonas aeruginosa promotes persistence of Stenotrophomonas maltophilia via increased adherence to depolarized respiratory epithelium

M. S. McDaniel, N. Lindgren, C. E. Billiot, K. N. Valladeres, N. A. Sumpter, W. E. Swords

This study shows Pseudomonas aeruginosa enhances Stenotrophomonas maltophilia persistence in CF lungs by disrupting epithelial barriers and promoting adherence via type IV pilus mechanisms.

Microbiology Spectrum · Feb 14, 2023Peer-reviewedMicrobiology

The Cu(II) reductase RclA protects Escherichia coli against the combination of hypochlorous acid and intracellular copper

R. M. Derke, A. J. Barron, C. E. Billiot, I. F. Chaple, S. E. Lapi, N. A. Broderick, M. J. Gray

This study shows that RclA, a Cu(II) reductase in E. coli, protects against HOCl and intracellular copper toxicity, highlighting copper redox reactions in inflammation.

bioRxiv · Jan 22, 2020Microbiology