Author

C. F.-d. Castillo

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Koch· 1 shared A. Liss· 1 shared A. Matsui· 1 shared D. Fukumura· 1 shared D. G. Duda· 1 shared D. H. Schanne· 1 shared G. Geidel· 1 shared H. Kikuchi· 1 shared

Activity by Year

2022-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

A. Koch

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1 paper

Recent·Most cited

IFNA pathway drives the more aggressive phenotype of KRASG12D-mutant pancreatic ductal adenocarcinomas via IFNAR1/STAT3 activation

D. G. Duda, K. Inoue, D. H. Schanne, A. Matsui, P. Lei, S. Klein, S. Aoki, H. Taniguchi, H. Kikuchi, J. Chen, Z. Liu, S. Q. Tsai, T. C. Schmidt, M. Iwasaki, G. Geidel, , N. Bardeesy

The paper shows that the IFNA pathway promotes aggressive KRASG12D-mutant pancreatic ductal adenocarcinomas through IFNAR1/STAT3 activation and identifies IFNAR1 inhibition as a potential therapeutic strategy.

bioRxiv · Jun 29, 2022 · PreprintCancer Biology