Author

C. Kamilaris

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Assay:proteomics· 1

Frequent Co-Authors

A. M. Lefrancois-Martinez· 2 shared A. Martinez· 2 shared D. Dufour· 2 shared I. Sahut-Barnola· 2 shared P. Val· 2 shared A. Levasseur· 1 shared A. Septier· 1 shared A. Swain· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersPathology

A. M. Lefrancois-Martinez

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2 papers

Recent·Most cited

Steroidogenic factor-1 lineage origin of skin lesions in Carney complex syndrome

I. Sahut-Barnola, A. M. Lefrancois-Martinez, D. Dufour, J.-M. Botto, C. Kamilaris, F. R. Faucz, C. A. Stratakis, P. Val, A. Martinez

This study identifies dermal fibroblasts as the origin of skin lesions in Carney complex by showing their pro-melanogenic activity under PKA signaling disruption.

bioRxiv · Jan 26, 2022Pathology

PKA drives paracrine crisis and WNT4-dependent testis tumor in Carney complex

C. Djari, I. Sahut-Barnola, A. Septier, I. Plotton, N. Montanier, D. Dufour, A. Levasseur, J. Wilmouth, J.-C. Pointud, F. Faucz, C. Kamilaris, A.-G. Lopez, F. Guillou, A. Swain, S. J. Vainio, , A. M. Lefrancois-Martinez

The paper investigates the role of PKA in Carney complex testis tumors, showing that Prkar1a mutations in both stromal and Sertoli cells drive LCCSCTs via WNT4-dependent paracrine signaling.

bioRxiv · Dec 21, 2020 · PreprintPathology