Author

C. McClenaghan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Assay:electrophysiology· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

A. Hanson· 2 shared C. G. Nichols· 2 shared D. K. Grange· 2 shared A. N. Stratman· 1 shared C. M. Halabi· 1 shared D. Josifova· 1 shared G. van Haaften· 1 shared H. I. Roessler· 1 shared

Activity by Year

2017-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersPhysiology

A. Hanson

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Author profile →

2 papers

Recent·Most cited

Electrophysiology of human iPSC-derived vascular smooth muscle cells and cell autonomous consequences of Cantu Syndrome mutations

A. Hanson, C. McClenaghan, K.-C. Weng, S. Colijn, A. N. Stratman, C. M. Halabi, D. K. Grange, J. R. Silva, C. G. Nichols

This study examines the electrophysiological properties of human iPSC-derived vascular smooth muscle cells and identifies cell-autonomous effects of Cantu Syndrome mutations on KATP channel function and vascular compliance.

bioRxiv · Jun 29, 2023Physiology

Cantu syndrome-associated SUR2 (ABCC9) mutations in distinct structural domains result in KATP channel gain-of-function by differential mechanisms

C. McClenaghan, A. Hanson, M. Sala-Rabanal, H. I. Roessler, D. Josifova, D. K. Grange, G. van Haaften, C. G. Nichols

This study investigates how specific SUR2 mutations in Cantu Syndrome alter KATP channel function through different mechanisms, with implications for drug response.

bioRxiv · Oct 26, 2017Biophysics