Author

C. M. Halabi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Hanson· 1 shared A. N. Stratman· 1 shared A. U. KABIR· 1 shared B. M. Brengle· 1 shared C. G. Nichols· 1 shared C. McClenaghan· 1 shared D. H. FREMONT· 1 shared D. K. Grange· 1 shared

Activity by Year

2022-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperPhysiology

A. Hanson

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Electrophysiology of human iPSC-derived vascular smooth muscle cells and cell autonomous consequences of Cantu Syndrome mutations

A. Hanson, C. McClenaghan, K.-C. Weng, S. Colijn, A. N. Stratman, C. M. Halabi, D. K. Grange, J. R. Silva, C. G. Nichols

This study examines the electrophysiological properties of human iPSC-derived vascular smooth muscle cells and identifies cell-autonomous effects of Cantu Syndrome mutations on KATP channel function and vascular compliance.

bioRxiv · Jun 29, 2023Physiology

Zbtb46 coordinates angiogenesis and immunity to control tumor outcome

A. U. KABIR, M. SUBRAMANIAN, M. KIM, J. WU, K. KRCHMA, X. WANG, C. M. HALABI, H. PAN, S. A. WICKLINE, D. H. FREMONT, K. CHOI

This study identifies Zbtb46 as a regulator that coordinates angiogenesis and immunity in tumors, with its downregulation promoting a pro-tumor microenvironment and its restoration improving tumor outcomes.

bioRxiv · Mar 2, 2023 · PreprintCancer Biology

Mutation in the matricellular gene fibulin-4 leads to endothelial dysfunction in resistance arteries

M. Lin, K. Jones, B. M. Brengle, R. P. Mecham, C. M. Halabi

This study shows that fibulin-4 mutations cause endothelial dysfunction in resistance arteries due to increased shear stress and oxidative environment.

bioRxiv · May 20, 2022 · PreprintPhysiology