Author

C. R. Ball

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Assay:ChIP-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1

Frequent Co-Authors

H. Glimm· 3 shared A. A. Wurm· 1 shared A. Banito· 1 shared A. Carazzato· 1 shared A. M. Dobberkau· 1 shared A. Nilsson· 1 shared A. Seiler· 1 shared B. C. Jones· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersCancer Biology

H. Glimm

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Author profile →

3 papers

Recent·Most cited

The histone modifier KAT2A presents a selective target in a subset of well-differentiated microsatellite-stable colorectal cancers

V. Kufrin, A. Seiler, S. Brilloff, H. Rothfuss, S. Schuster, S. Schaefer, E. Rahimian, J. Baumgarten, C. R. Ball, M. Bornhaeuser, H. Glimm, M. Bill, A. A. Wurm

This study identifies KAT2A as a therapeutic target in a subset of well-differentiated, microsatellite-stable colorectal cancers by showing its dependency and functional consequences.

bioRxiv · Nov 15, 2023Cancer Biology

Multidimensional Characterization of Soft-Tissue Sarcomas with FUS-TFCP2 or EWSR1-TFCP2 Fusions

J. Schopf, S. Uhrig, C. E. Heilig, K.-S. Lee, T. Walther, A. Carazzato, A. M. Dobberkau, D. Weichenhan, C. Plass, M. Hartmann, G. D. Diwan, Z. Carrero, C. R. Ball, T. Hohl, T. Kindler, , C. Scholl

This study characterizes soft-tissue sarcomas with FUS-TFCP2 or EWSR1-TFCP2 fusions through multi-omics analyses and functional studies, identifying therapeutic targets and mechanisms of disease.

bioRxiv · Feb 19, 2023Cancer Biology

YAP orchestrates heterotypic endothelial cell communication via HGF/c-MET signaling in liver tumorigenesis

S. Thomann, S. M. Weiler, S. Marquard, C. R. Ball, M. Toth, T. Wei, C. Sticht, C. de la Torre, E. Ryschich, O. Ermakova, C. Mogler, D. N. Kazdal, N. Gretz, H. Glimm, E. Rempel, , K. Breuhahn

This study shows YAP promotes liver tumor progression by altering the vascular niche, creating heterotypic communication between endothelial cell subtypes via HGF/c-MET signaling.

bioRxiv · Mar 17, 2020Cancer Biology