Author

G. D. Diwan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Evolutionary Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cell type annotation· 2 Organism:human· 2 Assay:proteomics· 2 Organism:bacteria (other)· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

R. B. Russell· 3 shared D. Agashe· 2 shared K. Boldt· 2 shared K. Junger· 2 shared A. Banito· 1 shared A. Carazzato· 1 shared A. M. Dobberkau· 1 shared A. Nilsson· 1 shared

Activity by Year

2018-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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3 shared papersCell Biology

R. B. Russell

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

DLG1 functions upstream of SDCCAG3 and IFT20 to control ciliary targeting of polycystin-2

C. K. Rezi, M. Aslanyan, G. D. Diwan, T. Cheng, M. Chamlali, K. Junger, Z. Anvarian, e. lorentzen, K. B. Pauly, Y. Bahadori, E. F. Fernandes, F. Qian, S. Tosi, S. T. Christensen, S. F. Pedersen, , L. B. Pedersen

This study shows DLG1 regulates ciliary protein trafficking in kidney cells, with loss of DLG1 causing ciliary elongation and cysts, and a specific DLG1 variant linked to kidney disease.

bioRxiv · Nov 10, 2023Cell Biology

Multidimensional Characterization of Soft-Tissue Sarcomas with FUS-TFCP2 or EWSR1-TFCP2 Fusions

J. Schopf, S. Uhrig, C. E. Heilig, K.-S. Lee, T. Walther, A. Carazzato, A. M. Dobberkau, D. Weichenhan, C. Plass, M. Hartmann, G. D. Diwan, Z. Carrero, C. R. Ball, T. Hohl, T. Kindler, , C. Scholl

This study characterizes soft-tissue sarcomas with FUS-TFCP2 or EWSR1-TFCP2 fusions through multi-omics analyses and functional studies, identifying therapeutic targets and mechanisms of disease.

bioRxiv · Feb 19, 2023Cancer Biology

Ciliopathy-associated missense mutations in IFT140 are hypomorphic and have edgetic effects on protein interaction networks

T. Leonhard, G. D. Diwan, F. Klose, I. F. Stehle, K. Junger, M. Seda, S. Bolz, F. Woerz, R. B. Russell, K. Boldt, D. Jenkins, M. Ueffing, T. Beyer

This study investigates how missense mutations in IFT140 affect protein interactions and complex stability, revealing hypomorphic and edgetic effects in ciliopathy-related diseases.

bioRxiv · Jan 18, 2023Cell Biology

Shifts in mutation spectra enhance access to beneficial mutations

M. Sane, G. D. Diwan, B. A. Bhat, L. M. Wahl, D. Agashe

The paper shows that changing mutation spectra in E. coli increases beneficial mutations and reduces deleterious load, with simulations indicating that bias shifts are selectively favored.

bioRxiv · Sep 5, 2020 · PreprintEvolutionary Biology

The Evolutionary History and Impact of Bacterial tRNA Modifications

G. D. Diwan, D. Agashe

This study reconstructs the evolutionary history of bacterial tRNA modifications and shows their role in shaping translation efficiency and tRNA gene repertoires.

bioRxiv · Jan 22, 2018Evolutionary Biology