Author

C. Wadsack

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Assay:flow cytometry· 1

Frequent Co-Authors

A. Heinemann· 2 shared J. Kargl· 2 shared O. Kindler· 2 shared Z. N. Mihalic· 2 shared A. Santiso· 1 shared G. Desoye· 1 shared G. Marsche· 1 shared I. Beceheli· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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2 shared papersCell Biology

A. Heinemann

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Myeloperoxidase enhances the migration of human choriocarcinoma JEG-3 cells

Z. N. Mihalic, T. Kloimboeck, N. Cosic-Mujkanovic, P. Valadez-Cosmes, K. Maitz, O. Kindler, C. Wadsack, A. Heinemann, G. Marsche, M. Gauster, J. Pollheimer, J. Kargl

This study shows that myeloperoxidase enhances migration of JEG-3 choriocarcinoma cells by internalization and activity, suggesting a role in EVT function during pregnancy.

bioRxiv · Jan 28, 2023 · PreprintCell Biology

Gestational diabetes mellitus dysregulates the PD-1/PD-L1 axis at the feto-maternal interface

Z. N. Mihalic, O. Kindler, S. Raftopoulou, A. Santiso, C. Wadsack, A. Heinemann, J. Kargl

The paper examines immune changes at the fetal-maternal interface in GDM, obesity, and healthy pregnancies, finding dysregulated PD-1/PD-L1 axis and reduced Treg infiltration in GDM.

bioRxiv · Jan 25, 2023 · PreprintImmunology

Differential serotonin uptake mechanisms at the human maternal-fetal interface

P. Bakovic, M. Kesic, M. Horvaticek, M. George, M. Peric, I. Beceheli, L. Cicin-Sain, G. Desoye, C. Wadsack, U. Panzenboeck, J. Stefulj

This study identifies distinct serotonin uptake mechanisms in human placental and fetal cells, showing high-affinity uptake in trophoblasts and low-affinity uptake in endothelial cells and platelets.

bioRxiv · Jan 7, 2021 · PreprintCell Biology