Author

Camille Glaser

Centre National de la Recherche Scientifique

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Biochemistry· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Aminah DawoodiD· 1 shared Anne HoudusseiD· 1 shared Debanjan BhowmikiD· 1 shared James A. SpudichiD· 1 shared Kathleen M. RuppeliD· 1 shared Matthew Carter ChildersiD· 1 shared Michael RegnieriD· 1 shared Neha NandwaniiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

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1 shared paperBiochemistry

Aminah DawoodORCID

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1 paper

Recent·Most cited

Hypertrophic cardiomyopathy mutations Y115H and E497D disrupt the folded-back state of human beta-cardiac myosin allosterically

Neha Nandwani, Debanjan Bhowmik, Camille Glaser, Matthew Carter Childers, Rama Reddy Goluguri, Aminah Dawood, Michael Regnier, Anne Houdusse, James A. Spudich, Kathleen M. Ruppel

This study investigates how HCM mutations Y115H and E497D disrupt the folded-back state of human beta-cardiac myosin, leading to hypercontractility by destabilizing the OFF-state.

Nature Communications · Oct 1, 2025Peer-reviewedBiochemistry