Author

D. H. Arabaci

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:mouse· 1

Frequent Co-Authors

A. Domaniku· 1 shared A. Loumaye· 1 shared B. Toledo· 1 shared B. Z. C. Weber· 1 shared J.-P. Thissen· 1 shared P. Lause· 1 shared S. Agca· 1 shared S. Kir· 1 shared

Activity by Year

2023-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

A. Domaniku

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1 paper

Recent·Most cited

EDA2R/NIK signaling promotes skeletal muscle atrophy linked to cancer cachexia

S. N. Bilgic, A. Domaniku, B. Toledo, S. Agca, B. Z. C. Weber, D. H. Arabaci, Z. Ozornek, P. Lause, J.-P. Thissen, A. Loumaye, S. Kir

This study identifies EDA2R/NIK signaling as a driver of cancer cachexia-induced skeletal muscle atrophy and shows that targeting this pathway can prevent muscle loss.

bioRxiv · Jan 23, 2023Cancer Biology