Author

D. H. Kaplan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:proteomics· 1

Frequent Co-Authors

s. gaffen· 2 shared T. Edwards· 2 shared A. Liu· 1 shared a. mathers· 1 shared b. davis· 1 shared b. hube· 1 shared c. d'enfert· 1 shared C. Zhou· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersImmunology

s. gaffen

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2 papers

Recent·Most cited

A loss-of-function mutation in IL-17F enhances susceptibility of mice to oropharyngeal candidiasis

C. Zhou, L. Monin, R. Gordon, F. E. Aggor, R. Bechara, T. Edwards, D. H. Kaplan, S. Gingras, s. gaffen

This study shows that a loss-of-function mutation in IL-17F increases susceptibility to oropharyngeal candidiasis in mice, but not in heterozygous carriers.

bioRxiv · Apr 9, 2020Immunology

Extracellular ATP released from Candida albicans activates non-peptidergic neurons to augment host defense

T. Edwards, S. Zhang, A. Liu, J. Cohen, P. Zhou, S. Mogavero, b. hube, j. berman, m.-e. bougnoux, a. mathers, s. gaffen, K. M. Albers, h. r. koeber, b. davis, c. d'enfert, D. H. Kaplan

This study shows that ATP released by Candida albicans activates non-peptidergic neurons to enhance anti-Candida immune responses.

bioRxiv · Jan 27, 2020 · PreprintImmunology