Author

E. Bloom-Saldana

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 3

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Research Focus:differential expression· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 1 Assay:metagenomics· 1 Approach:statistical model· 1

Frequent Co-Authors

J. M. Irimia· 3 shared P. T. Fueger· 3 shared B. M. Bauer· 2 shared A. Castillo· 1 shared E. L. Cauble· 1 shared J. C. McDonough· 1 shared J.-W. Jeong· 1 shared L. S. Trevino· 1 shared

Activity by Year

2023-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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3 shared papersPhysiology

J. M. Irimia

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Longitudinal analysis of a dietary mouse model of non-alcoholic fatty liver disease (NAFLD) and non-alcoholic steatohepatitis (NASH)

M. M. Saenz, J. C. McDonough, E. Bloom-Saldana, J. M. Irimia, E. L. Cauble, A. Castillo, P. T. Fueger, L. S. Trevino

This study tracks histopathological, biochemical, transcriptomic, and microbiome changes in mice fed a NASH-promoting diet over 30 weeks, revealing progressive NAFLD development and immune/xenobiotic gene expression changes.

bioRxiv · May 19, 2023 · PreprintPhysiology

Dose-dependent progression of multiple low dose streptozotocin-induced diabetes in mice

B. M. Bauer, S. Bhattacharya, E. Bloom-Saldana, J. M. Irimia, P. T. Fueger

This study compares the effects of two low-dose streptozotocin regimens in mice, showing distinct glucose tolerance, beta cell function, and transcriptional changes.

bioRxiv · Apr 8, 2023Physiology

Pancreatic loss of Mig6 alters murine endocrine cell fate and protects functional beta cell mass in an STZ-induced model of diabetes

B. M. Bauer, J. M. Irimia, E. Bloom-Saldana, J.-W. Jeong, P. T. Fueger

This study shows that pancreatic loss of Mig6 increases beta cell mass and improves glucose tolerance in a diabetes model, but beta cell-specific loss of Mig6 does not provide similar protection.

bioRxiv · Apr 7, 2023 · PreprintPhysiology