Zinc dysregulation in slc30a8 (znt8) mutant zebrafish leads to blindness and disrupts bone mineralisation
E. W. Mackay, S. Ibanez Molero, L. Harjani Tirathdas, J. Peterson-Maduro, J. Zang, S. C. Neuhauss, S. Schulte-Merker, S. W. Wilson
This study shows that slc30a8 mutants in zebrafish develop blindness and bone mineral defects due to zinc accumulation in non-pigmented tissues, suggesting a role for pigment cells in zinc homeostasis.
bioRxiv · Sep 2, 2020 · PreprintPhysiology