PhysiologybioRxivPreprint
Zinc dysregulation in slc30a8 (znt8) mutant zebrafish leads to blindness and disrupts bone mineralisation
E. W. Mackay, S. Ibanez Molero, L. Harjani Tirathdas…·S. W. Wilson
This study shows that slc30a8 mutants in zebrafish develop blindness and bone mineral defects due to zinc accumulation in non-pigmented tissues, suggesting a role for pigment cells in zinc homeostasis.