Author

F. Coulon

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Bioengineering· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Organism:human· 1 Organism:mouse· 1 Research Focus:variant calling· 1

Frequent Co-Authors

F. Halary· 2 shared R. Josien· 2 shared A. Even· 1 shared A. Garcia· 1 shared A. Molle· 1 shared A. Moreau· 1 shared A. Touze· 1 shared C. Braudeau· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersBioengineering

F. Halary

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Controlling fibroblast fibrinolytic activity allows for the bio-engineering of stable connective tissue equivalents

E. Boucard, F. Coulon, L. Vidal, J.-Y. Hascoet, C. Mota, F. Halary

The paper shows that controlling fibroblast fibrinolytic activity can stabilize GAF-based biomaterials by inhibiting MMPs and urokinase.

bioRxiv · Sep 21, 2021 · PreprintBioengineering

Non-permissive human conventional CD1c+ dendritic cells enable trans-infection of human primary renal tubular epithelial cells and protect BK polyomavirus from neutralization

M. Sikorski, F. Coulon, C. Peltier, C. Braudeau, A. Garcia, M. Giraud, K. Autain-Renaudin, C. Kandel-Aznar, S. Nedellec, P. Hulin, J. Branchereau, J. Veziers, P. Gaboriaud, A. Touze, J. Burlaud-Gaillard, , F. Halary

This study shows that CD1c+ dendritic cells can transmit BKPyV to epithelial cells and protect the virus from neutralization.

bioRxiv · Sep 4, 2020Microbiology

Dendritic cells require TMEM176A/B ion channels for optimal MHC II antigen presentation to naive CD4+ T cells

M. Lancien, G. Bienvenu, L. Gueno, S. Salle, E. Merieau, S. Remy, A. Even, A. Moreau, A. Molle, C. Fourgeux, F. Coulon, G. Beriou, L. Bouchet-Delbos, E. Chiffoleau, P. Kirstetter, , C. Louvet

The paper shows TMEM176A/B ion channels are essential for dendritic cells to present antigens to CD4+ T cells, but not for Th17 or ILC3 function.

bioRxiv · May 1, 2020 · PreprintImmunology