Author

G. J. M. Cowan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Assay:scRNA-seq· 3 Organism:human· 2 Approach:statistical model· 2 Research Focus:clustering· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:machine learning (classical)· 1

Frequent Co-Authors

C. Goodyear· 2 shared C. Sutherland· 2 shared D. Gray· 2 shared K. Miles· 2 shared M. Gray· 2 shared S. Breusch· 2 shared E. Wing· 1 shared H. Johnsson· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersImmunology

C. Goodyear

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3 papers

Recent·Most cited

ImmunologybioRxivPreprintFeb 15, 2023

A comprehensive analysis of rheumatoid arthritis B cells reveals the importance of CD11c+ve double-negative-2 B cells as the major synovial plasma cell precursor.

E. Wing, C. Sutherland, K. Miles…·M. Gray

This study identifies CD11c+ve double-negative-2 B cells as the major synovial plasma cell precursor in rheumatoid arthritis using single-cell RNA and repertoire sequencing.

DOI 10.1101/2023.02.15.526468

humanscRNA-seq

𝕏 PostBluesky

ImmunologybioRxivJul 27, 2020

A Conserved TCRβ signature dominates a highly polyclonal T-cell expansion during the acute phase of a murine malaria infection

N. L. Smith, W. Nahrendorf, C. Sutherland…·G. J. M. Cowan

The paper identifies a conserved TCRβ signature dominated by TRBV3 gene usage in a highly polyclonal T-cell expansion during the acute phase of a murine malaria infection.

DOI 10.3389/fimmu.2020.587756

mousescRNA-seq

𝕏 PostBluesky

ImmunologybioRxivPreprintSep 18, 2019

Rheumatoid arthritis patients express a skewed repertoire of polyclonal, hypomutated B-cell receptors

G. J. M. Cowan, K. Miles, L. Capitani…·M. Gray

This study identifies that RA patients have a higher proportion of poorly mutated, polyclonal IgG+ve BCRs in peripheral blood and synovium, suggesting a role in autoimmune inflammation.

DOI 10.1101/771949

humanscRNA-seq

𝕏 PostBluesky