Author

G. Zito

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Mangogna· 2 shared A. Martorana· 2 shared B. Belmonte· 2 shared F. Romano· 2 shared G. Ricci· 2 shared A. Balduit· 1 shared A. Giorgiutti· 1 shared A. Romano· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersCell Biology

A. Mangogna

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2 papers

Recent·Most cited

PARP-1, EpCAM, and FRα as potential targets for intraoperative detection and delineation of endometriosis: a quantitative tissue expression analysis

B. Belmonte, G. Di Lorenzo, A. Mangogna, B. Bortot, G. Bertolazzi, S. Sammataro, S. Merighi, A. Martorana, G. Zito, F. Romano, A. Giorgiutti, C. Bottin, F. Zanconati, A. Romano, G. Ricci, S. Biffi

This study evaluates PARP-1, EpCAM, and FR as potential biomarkers for intraoperative detection of endometriosis through gene expression and immunohistochemical analysis.

bioRxiv · Dec 19, 2023Cell Biology

Complement Component 3 expressed by the endometrial ectopic tissue is involved in the endometriotic lesion formation through mast cell activation

C. Agostinis, S. Zorzet, A. Balduit, G. Zito, A. Mangogna, P. Macor, F. Romano, M. Toffoli, B. Belmonte, A. Martorana, V. Borelli, G. Ricci, U. Kishore, R. Bulla

This study shows that complement component 3 from endometrial ectopic tissue promotes endometriotic lesion formation by activating mast cells in a murine model.

bioRxiv · Nov 19, 2020 · PreprintImmunology