Author

H. Inam

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Evolutionary Biology· 1

Signature Assays, Organisms & Methods

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Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1

Frequent Co-Authors

J. R. Pritchard· 2 shared A. Shah· 1 shared B. Zhao· 1 shared C. Dong· 1 shared C. Liu· 1 shared D. M. McCandlish· 1 shared E. O'Brien· 1 shared F. Naeemikia· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersBioengineering

J. R. Pritchard

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2 papers

Recent·Most cited

Genomic and experimental evidence that alternate transcription initiation of the Anaplastic Lymphoma Kinase (ALK) kinase domain does not predict single agent sensitivity to ALK inhibitors.

H. Inam, I. Sokirniy, Y. Rao, A. Shah, F. Naeemikia, E. O'Brien, C. Dong, D. M. McCandlish, J. R. Pritchard

The paper reanalyzes ALKATI's role in melanoma using genomic and experimental methods, finding it does not predict single-agent ALK inhibitor sensitivity.

bioRxiv · Jun 17, 2021 · PreprintBioengineering

Multi-scale predictions of drug resistance epidemiology suggest a new design principle for rational drug design.

S. Leighow, C. Liu, H. Inam, B. Zhao, J. R. Pritchard

This paper identifies a new principle in drug resistance where the most likely mutations, not the most resistant ones, dominate clinical outcomes, suggesting a strategy to minimize resistance through drug design.

bioRxiv · Sep 19, 2019 · PreprintEvolutionary Biology