Author

H. Stachelscheid

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Organism:human· 2 Approach:deep learning· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Assay:organoid / primary culture· 1

Frequent Co-Authors

C. Scharler· 2 shared D. STRUNK· 2 shared K. Schallmoser· 2 shared M. Wolf· 2 shared P. Peking· 2 shared R. Poupardin· 2 shared A. Hoog· 1 shared A. Schneeberger· 1 shared

Activity by Year

2020-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

C. Scharler

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Self-assembly of progenitor cells under the aegis of platelet factors facilitates human skin organoid formation and vascularized wound healing

P. Peking, L. Krisch, M. Wolf, A. Hoog, B. Vari, R. Poupardin, C. Scharler, L. Russe, H. Stachelscheid, A. Schneeberger, K. Schallmoser, D. STRUNK

This study shows that platelet factors enable the self-assembly of skin cells into organoids and accelerate wound healing in mice.

bioRxiv · Sep 10, 2020 · PreprintCell Biology

Extra-hematopoietic immunomodulatory role of the SCID-susceptibility gene DOCK-2 identified by stepwise maturation of human iPSCs into clonogenic mesodermal stromal progenitors

C. Scharler, R. Poupardin, P. Peking, M. Wolf, G. Brachtl, L. Daheron, K. Schallmoser, K. Juerchott, H. Stachelscheid, H.-D. Volk, D. STRUNK

This study identifies DOCK-2's role in stromal immunomodulation through iPSC differentiation and CRISPR knockout, showing reduced immunomodulatory capacity in DOCK-2-deficient cells.

bioRxiv · Jul 7, 2020 · PreprintImmunology