Author

H. Winata

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:bulk RNA-seq· 1 Approach:deep learning· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

C. Nian· 2 shared F. C. Lynn· 2 shared S. Sasaki· 2 shared D. J. Pasula· 1 shared D. S. Luciani· 1 shared E. E. Xu· 1 shared J. S. Yoon· 1 shared J. Velghe· 1 shared

Activity by Year

2023-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersPhysiology

C. Nian

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2 papers

Recent·Most cited

Calcium-dependent transcriptional profiles of human pancreatic islet cells reveal functional diversity in islet subpopulations

J. S. Yoon, S. Sasaki, J. Velghe, K. Zosel, M. Y. Y. Lee, H. Winata, C. Nian, F. C. Lynn

The paper profiles calcium-regulated gene expression in human pancreatic islet cells using single-cell RNA sequencing and identifies PCDH7 as a novel beta cell marker with enhanced insulin secretory function.

bioRxiv · Sep 8, 2023 · PreprintPhysiology

Type 2 diabetes susceptibility gene GRK5 regulates physiological pancreatic β-cell proliferation via phosphorylation of HDAC5 in mice

S. Sasaki, C. Nian, E. E. Xu, D. J. Pasula, H. Winata, S. Grover, D. S. Luciani, F. C. Lynn

This study identifies GRK5 as a regulator of pancreatic β-cell proliferation through phosphorylation of HDAC5, linking it to type 2 diabetes pathogenesis.

iScience · Aug 1, 2023Peer-reviewedPhysiology