Author

H.-L. Ji

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pharmacology & Toxicology· 2 Cell Biology· 1 Microbiology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cell type annotation· 3 Organism:human· 3 Organism:mouse· 3 Assay:proteomics· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:differential expression· 2 Approach:biophysical modeling· 1

Frequent Co-Authors

R. Zhao· 4 shared G. Ali· 2 shared H.-G. Nie· 2 shared J. Chang· 2 shared M. A. Matthay· 2 shared A. W. Tao· 1 shared B. M. Samten· 1 shared B. Zhou· 1 shared

Activity by Year

2020-2024 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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4 shared papersPharmacology & Toxicology

R. Zhao

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Frequent intertrophic transmission of Wolbachia by parasitism but not predation

Z.-C. Yan, L.-D. Qi, H.-L. Ji, X.-X. Wang, X.-Y. Hong, Y.-X. Li

This study identifies parasitism as the main route of intertropical Wolbachia transmission, showing evidence of bidirectional transfers between parasitoid wasps and their hosts.

bioRxiv · Mar 20, 2024 · PreprintMicrobiology

Regenerative Signatures in Bronchioalveolar Lavage of Acute Respiratory Distress Syndrome

R. Zhao, M. Hadisurya, H. Ndetan, N. M. Xi, S. Adduri, N. V. Konduru, B. M. Samten, A. W. Tao, K. P. Singh, H.-L. Ji

This study identifies differentially expressed exosomal proteins in ARDS patients that may serve as regenerative niches for lung recovery.

bioRxiv · Nov 13, 2023 · PreprintPathology

Ion transport mechanisms for smoke inhalation injured airway epithelial barrier

J. Chang, Z. Chen, R. Zhao, H.-G. Nie, H.-L. Ji

This study investigates how thermal stress and acrolein, a smoke toxin, affect airway epithelial barrier function by altering ion transport pathways and tight junction integrity.

bioRxiv · Mar 25, 2020Pharmacology & Toxicology

Fibrinolytic niche is requested for alveolar type 2 cell-mediated alveologenesis and injury repair

G. Ali, R. Zhao, M. Zhang, K. G. Jain, J. Chang, S. Komatsu, X. Fang, B. Zhou, J. Liang, D. Jiang, M. Ikebe, M. A. Matthay, H.-L. Ji

This study shows that the fibrinolytic niche regulates alveolar type 2 cell function and regeneration through the uPA-A6-CD44-ENaC cascade, with implications for lung injury repair.

bioRxiv · Mar 24, 2020 · PreprintCell Biology

Plasmin improves oedematous blood-gas barrier by cleaving epithelial sodium channels

R. Zhao, G. Ali, H.-G. Nie, Y. Chang, D. Bhattarai, X. Su, X. Zhao, M. A. Matthay, H.-L. Ji

This study shows plasmin cleaves ENaC channels at multiple sites, enhancing fluid clearance in lung injury models.

bioRxiv · Feb 9, 2020Pharmacology & Toxicology