Author

I. B. Kilic

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Kandilci· 2 shared T. Guzel· 2 shared A. Avci· 1 shared A. Kalayci· 1 shared A. Karahasan· 1 shared A. Yasar· 1 shared B. Celik· 1 shared D. Yoleri· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersImmunology

A. Kandilci

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

NETosis Induced by Serum of Patients with COVID-19 is Reduced with Reparixin or Antibodies Against DEK and IL-8

I. B. Kilic, A. Yasar, I. Yalim Camci, T. Guzel, A. Karahasan, T. Yagci, N. Cine, A. Kandilci

This study shows that reparixin or anti-DEK antibodies reduce NETosis induced by COVID-19 patient serum, suggesting DEK-CXCR2 interaction drives this process.

bioRxiv · Mar 30, 2023 · PreprintImmunology

Doxorubicin induces prolonged DNA damage signal in cells overexpressing DEK isoform-2.

E. Ozcelik, A. Kalayci, B. Celik, A. Avci, H. Akyol, I. B. Kilic, T. Guzel, M. Cetin, M. Tuzlakoglu Ozturk, Z. O. Caliskaner, M. Tombaz, D. Yoleri, O. Konu, A. Kandilci

This study shows that DEK isoform-2 induces prolonged DNA damage signals and is functionally distinct from DEK isoform-1 in response to doxorubicin.

bioRxiv · Jun 3, 2022 · PreprintCell Biology