Author

J. A. Ellis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

A. Mahady· 1 shared E. T. Wang· 1 shared J. A. Berglund· 1 shared J. M. Holaska· 1 shared J. Richardson· 1 shared K. A. Bossone· 1 shared M. Hale· 1 shared O. McConnell· 1 shared

Activity by Year

2020-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperBiochemistry

A. Mahady

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2 papers

Recent·Most cited

MBNL1 and RBFOX1 co-regulate alternative splicing events transcriptome-wide through a conserved buffering mechanism

J. A. Berglund, M. Hale, J. A. Ellis, R. Meng, S. McDaniel, A. Mahady, S. Wagner, J. Richardson, O. McConnell, E. T. Wang

This study identifies a conserved buffering mechanism where MBNL1 and RBFOX1 co-regulate alternative splicing by overlapping RNA motifs, reducing the range of splicing outcomes.

bioRxiv · Apr 20, 2021 · PreprintBiochemistry

Histone acetyltransferase inhibition rescues differentiation of emerin-null myogenic progenitors

K. A. Bossone, J. A. Ellis, J. M. Holaska

The paper shows that inhibiting histone acetyltransferases rescues differentiation defects in emerin-null myogenic progenitors, suggesting H4K5 acetylation dynamics are critical for myogenic differentiation in EDMD1.

bioRxiv · Feb 25, 2020Cell Biology