Author

J. Erenpreisa

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Research Focus:chromatin & epigenetic regulation· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

B. I. Gerashchenko· 2 shared D. Pjanova· 2 shared K. Salmina· 2 shared M. Lazovska· 2 shared A. Giuliani· 1 shared G. Zimatore· 1 shared K. Yoshikawa· 1 shared M. S. Cragg· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

B. I. Gerashchenko

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Liver regeneration by oval cells employing bistability of stemness-senescence, Hippo signaling, EMT-MET, and polyploidy circuit

M. Lazovska, K. Salmina, D. Pjanova, B. I. Gerashchenko, J. Erenpreisa

This paper investigates liver regeneration by oval cells, revealing a bistable switch between stemness and senescence regulated by Hippo signaling, EMT-MET, and polyploidy circuits.

bioRxiv · Mar 26, 2024 · PreprintCell Biology

Role of the circadian clock "Death-Loop" in the DNA damage response underpinning cancer treatment resistance

N. M. Vainshelbaum, K. Salmina, B. I. Gerashchenko, M. Lazovska, P. Zayakin, M. S. Cragg, D. Pjanova, J. Erenpreisa

The paper investigates how the circadian clock 'death-loop' contributes to cancer treatment resistance by enabling DNA damage tolerance and telomere renewal in polyploid cancer cells.

bioRxiv · Jan 14, 2022Cancer Biology

A Unified Genomic Mechanism of Cell-Fate Change

M. Tsuchiya, A. Giuliani, G. Zimatore, J. Erenpreisa, K. Yoshikawa

The paper proposes a genomic mechanism explaining cell-fate change through critical points and attractor dynamics in genome expression.

bioRxiv · Nov 24, 2021 · PreprintCell Biology