Author

J. Forejt

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Evolutionary Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Assay:whole-genome sequencing· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

P. Jansa· 2 shared A. Bondarieva· 1 shared A. Bryjova· 1 shared A. Orth· 1 shared A. Schleiffer· 1 shared A. Toth· 1 shared B. de Massy· 1 shared B. V. Bimova· 1 shared

Activity by Year

2017-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersCell Biology

P. Jansa

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Phenogenomic resources immortalized in a panel of wild-derived strains of five species of house mice

J. Pialek, L. Dureje, Z. Hiadlovska, J. Kreisinger, T. Aghova, A. Bryjova, D. Cizkova, J. Gouy de Bellocq, H. Hejlova, K. Janotova, I. Martincova, A. Orth, J. Pialkova, I. Pospisilova, L. Rouskova, , P. Klusackova

This study compares genetic and phenotypic data of wild-derived mouse strains with classical laboratory strains, finding greater genetic diversity in wild strains and phenotypic differences in multiple traits.

bioRxiv · Nov 5, 2023Evolutionary Biology

ANKRD31 regulates spatiotemporal patterning of meiotic recombination initiation and ensures recombination between heterologous sex chromosomes in mice

F. Papanikos, J. A. J. Clement, E. Testa, R. Ravindranathan, C. Grey, I. Dereli, A. Bondarieva, S. Valerio-Cabrera, M. Stanzione, A. Schleiffer, P. Jansa, D. Lustyk, F. Jifeng, J. Forejt, M. Barchi, , A. Toth

This study identifies ANKRD31 as essential for meiotic recombination timing and spatial organization, particularly in sex chromosomes, leading to segregation failure in its absence.

bioRxiv · Sep 28, 2018Cell Biology

Modulation of Prdm9-controlled meiotic chromosome asynapsis overrides hybrid sterility in mice

S. Gregorova, V. Gergelits, I. Chvatalova, T. Bhattacharyya, B. Valiskova, V. Fotopulosova, P. Jansa, D. Wiatrowska, J. Forejt

The paper investigates how Prdm9-controlled meiotic asynapsis contributes to hybrid sterility in mice and shows that introducing consubspecific homology can restore synapsis and fertility.

bioRxiv · Dec 11, 2017Evolutionary Biology