Author

J. Kallenbach

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Assay:imaging· 1 Approach:statistical model· 1

Frequent Co-Authors

A. E. Loiselle· 1 shared A. Grosberg· 1 shared A. Harriot· 1 shared A. van Wijnen· 1 shared C. Venegas· 1 shared C. Ward· 1 shared D. Abplanalp· 1 shared G. Shi· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

A. E. Loiselle

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Author profile →

2 papers

Recent·Most cited

Myofibrillar malformations that arise in mdx muscle fibers are driven by detyrosinated microtubules

A. Harriot, T. Altair-Morris, C. Venegas, J. Kallenbach, K. Pinto, H. Joca, M.-J. Moutin, G. Shi, J. Ursitti, A. Grosberg, C. Ward

This study identifies detyrosinated microtubules as a driver of myofibrillar malformations in dystrophic skeletal muscle fibers, linking increased deTyr-MT levels to structural impairments.

bioRxiv · Mar 27, 2023 · PreprintPathology

Altered TGFB1 regulated pathways promote accelerated tendon healing in the superhealer MRL/MpJ mouse

J. Kallenbach, M. Freeberg, D. Abplanalp, J. Lillis, J. Ashton, A. E. Loiselle, M. Buckley, A. van Wijnen, H. A. Awad

The study compares tendon healing in MRL/MpJ and C57Bl/6J mice, finding altered TGFB1 pathways in MRL mice that reduce fibrosis and promote faster healing.

bioRxiv · Feb 8, 2021Cell Biology