Author

Jane Vowles

Primary ORCID profile (0000-0002-7000-1581 · 4 papers)Unclaimed ORCID profile · follow to get notified of new preprints

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Same name, identified by ORCID

Jane VowlesiD4 papers · University of Oxford

Biology Disciplines

Click to filter author papers

Microbiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

Alun Vaughan-Jackson· 1 shared Henry Owen· 1 shared Lea Nussbaum· 1 shared M.D. MooreiD· 1 shared William James· 1 shared

Activity by Year

2018-2018 · peak 1/yr

Co-Author Collaboration Network

5 top collaborators · 10 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperMicrobiology

Alun Vaughan-Jackson

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

HIV-1 evasion of restriction factors: cyclophilin A and cell fusion provide a helping hand.

Henry Owen, Alun Vaughan-Jackson, Lea Nussbaum, Jane Vowles, William James, M.D. Moore

This study shows TRIMCyp restricts HIV-1 cell-free infection, while cell-cell fusion between macrophages and T cells creates 'heterocytia' that evade multiple restriction factors and AZT.

bioRxiv · Aug 2, 2018 · PreprintMicrobiology