Author

Jennifer Lachowiec

Primary ORCID profile (0000-0003-2962-6448 · 2 papers)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

Jennifer LachowieciD2 papers · Montana State University

Biology Disciplines

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Evolutionary Biology· 1 Genetics· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Approach:statistical model· 3 Organism:Arabidopsis· 2 Research Focus:gene regulatory network inference· 2 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Christine Queitsch· 2 shared Christine QueitschiD· 1 shared Elhanan Borenstein· 1 shared G. Alex Mason· 1 shared Karla Schultz· 1 shared Örjan CarlborgiD· 1 shared Tzitziki Lemus· 1 shared Xia Shen· 1 shared

Activity by Year

2014-2016 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 9 mutual co-author links

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2 shared papersGenetics

Christine Queitsch

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3 papers

Recent·Most cited

Redundancy, feedback, and robustness in the Arabidopsis thaliana BZR/BEH gene family

Jennifer Lachowiec, G. Alex Mason, Karla Schultz, Christine Queitsch

The paper investigates the role of the Arabidopsis thaliana BZR/BEH gene family in developmental robustness, finding that only BEH4 contributes to trait robustness, challenging assumptions about functional redundancy.

bioRxiv · May 14, 2016 · PreprintGenetics

A Genome-Wide Association Analysis Reveals Epistatic Cancellation of Additive Genetic Variance for Root Length in Arabidopsis thaliana

Jennifer Lachowiec, Xia Shen, Christine Queitsch, Örjan Carlborg

This paper identifies epistatic genetic interactions underlying root length variation in Arabidopsis thaliana, revealing that epistasis cancels additive genetic variance, and discovers functional candidate genes.

PLOS Genetics · Sep 23, 2015Peer-reviewedGenomics

Hsp90 promotes kinase evolution

Jennifer Lachowiec, Tzitziki Lemus, Elhanan Borenstein, Christine Queitsch

This study shows Hsp90 client status increases kinase evolutionary rate and genetic variation, supporting the capacitor hypothesis, but client status is more stable than expected across kinases.

Molecular Biology and Evolution · Sep 21, 2014Peer-reviewedEvolutionary Biology