Author

Jonathan N. Ouellette

University of Wisconsin–Madison

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Organism:mouse· 1 Organism:rat· 1 Approach:statistical model· 1

Frequent Co-Authors

Andrea C. Ewald· 2 shared Avtar RoopraiD· 2 shared Jyoti J. WattersiD· 2 shared Abigail B. Radcliff· 1 shared Abiye Agbeh· 1 shared Andrew O. KnutsoniD· 1 shared Armand L. Meza· 1 shared Bailey A. KermathiD· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersNeuroscience

Andrea C. Ewald

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2 papers

Recent·Most cited

Recurrent hypoxia in a rat model of sleep apnea during pregnancy leads to microglia-dependent respiratory deficits and persistent neuroinflammation in adult male offspring

Carly R. Mickelson, Andrea C. Ewald, Maia G. Gumnit, Armand L. Meza, Abigail B. Radcliff, Stephen Mark Johnson, Jonathan N. Ouellette, Bailey A. Kermath, Avtar Roopra, Michael E. Cahill, Jyoti J. Watters, Tracy L. Baker

This study shows that maternal sleep apnea during pregnancy in rats leads to long-term respiratory deficits and neuroinflammation in male offspring due to microglia changes, while female offspring are unaffected.

bioRxiv · Dec 20, 2022 · PreprintNeuroscience

Prior hypoxia exposure enhances murine microglial inflammatory gene expression in vitro without concomitant H3K4me3 enrichment

Elizabeth A. Kiernan, Andrea C. Ewald, Jonathan N. Ouellette, Tao Wang, Abiye Agbeh, Andrew O. Knutson, Avtar Roopra, Jyoti J. Watters

This study shows that prior hypoxia exposure increases microglial inflammatory gene expression in response to LPS without increasing H3K4me3 at these genes.

Frontiers in Cellular Neuroscience · Oct 7, 2020Peer-reviewedNeuroscience