Author

M Schmelz

Heidelberg University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Cell Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

BS Sahu· 1 shared G Suresh· 1 shared H Chakravarthy· 1 shared I Kaur· 1 shared KK Bali· 1 shared S Sharma· 1 shared S Shree· 1 shared Sushma Dagar· 1 shared

Activity by Year

2026-2026 · peak 1/yr

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9 top collaborators · 36 mutual co-author links

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1 shared paperCell Biology

BS Sahu

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1 paper

Recent·Most cited

Hyperglycemia transcriptionally regulates the paranodal protein (Caspr1) in retinal neurons and modulates neurite extension

S Sharma, S Shree, G Suresh, H Chakravarthy, M Schmelz, KK Bali, Sushma Dagar, BS Sahu, I Kaur, Vasudharani Devanathan

This study shows hyperglycemia reduces Caspr1 expression in retinal neurons via Akt and C/EBP pathways, promoting neurite outgrowth, while insulin treatment under hyperglycemia decreases Caspr1 and shortens neurites.

bioRxiv · Jan 3, 2026 · PreprintCell Biology