Author

M. C. Essers

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 4

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 4 Research Focus:cell type annotation· 3 Assay:electrophysiology· 3 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1

Frequent Co-Authors

H. Abriel· 4 shared J.-S. Rougier· 3 shared L. Gillet· 2 shared P. Arullampalam· 2 shared S. Guichard· 2 shared D. Shmerling· 1 shared J. S. Rougier· 1 shared L. C. Ozhathil· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

9 top collaborators · 22 mutual co-author links

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4 shared papersPhysiology

H. Abriel

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4 papers

Recent·Most cited

Knockdown of the TRPM4 channel alters cardiac electrophysiology and hemodynamics in a sex- and age-dependent manner in mice

P. Arullampalam, M. C. Essers, J. S. Rougier, H. Abriel

This study shows that TRPM4 knockdown in mice affects cardiac electrophysiology and hemodynamics differently in males and females, with effects becoming apparent in adulthood.

Physiological Reports · Aug 21, 2023Peer-reviewedPhysiology

Deletion of Trpm4 alters the function and expression of NaV1.5 channel in murine cardiac myocytes

L. C. Ozhathil, J.-S. Rougier, P. Arullampalam, M. C. Essers, H. Abriel

This study shows that Trpm4 deletion in mice reduces NaV1.5 expression and Na+ currents in cardiac myocytes, leading to altered electrophysiological properties.

bioRxiv · Jul 6, 2020Physiology

A distinct pool of Nav1.5 channels at the lateral membrane of murine ventricular cardiomyocytes

J.-S. Rougier, M. C. Essers, L. Gillet, S. Guichard, S. Sonntag, D. Shmerling, H. Abriel

This study investigates the role of 1-syntrophin in regulating sodium current at the lateral membrane of cardiomyocytes and identifies a sub-pool of Nav1.5 channels independent of 1-syntrophin and the PDZ-binding motif.

bioRxiv · Mar 9, 2019Physiology

Dystrophin and calcium current are decreased in cardiomyocytes expressing Cre enzyme driven by αMHC but not TNT promoter

L. Gillet, S. Guichard, M. C. Essers, J.-S. Rougier, H. Abriel

This study shows that MHC-Cre mice exhibit reduced dystrophin and calcium current, unlike TNT-Cre mice, highlighting the need for Cre-only controls in experiments.

bioRxiv · Feb 1, 2019Physiology