Author

M. Chaudhuri

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 1 Organism:human· 1 Organism:mouse· 1 Organism:multiple species· 1 Assay:proteomics· 1 Organism:rat· 1

Frequent Co-Authors

A. Cooley· 2 shared A. Tripathi· 2 shared C. Darden· 2 shared J. Davis· 2 shared E. Krystofiak· 1 shared F. S. Gonzalez· 1 shared J. E. Donkor· 1 shared J. Smith· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 32 mutual co-author links

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2 shared papersCell Biology

A. Cooley

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Distinct structural motifs are necessary for targeting and import of Tim17 in Trypanosoma brucei mitochondrion

C. Darden, J. E. Donkor, O. Korolkova, M. Y. Khan Barozai, M. Chaudhuri

The paper identifies internal mitochondrial targeting signals and a critical residue in TbTim17 necessary for its import and integration into the mitochondrion of Trypanosoma brucei.

bioRxiv · Jul 7, 2023 · PreprintCell Biology

Unique interactions and functions of the mitochondrial small Tims in Trypanosoma brucei

L. S. Quinones Guillen, F. S. Gonzalez, C. Darden, M. Khan, A. Tripathi, J. Smith, A. Cooley, V. Paromov, J. Davis, S. Misra, M. Chaudhuri

This study identifies unique interactions and functional roles of small TbTims in the TbTIM17 complex of T. brucei, revealing distinct complex architectures and dynamics.

bioRxiv · May 29, 2023 · PreprintCell Biology

Trypanosoma brucei Tim50 Plays a Critical Role in Cell Cycle Regulation and Parasite Infectivity

A. Tripathi, U. K. Singha, A. Cooley, T. Gillyard, E. Krystofiak, S. Pratap, J. Davis, M. Chaudhuri

This study shows that TbTim50 is essential for mitochondrial function, kDNA replication, and cell cycle regulation in Trypanosoma brucei, and its depletion reduces parasite infectivity.

bioRxiv · Apr 26, 2021Cell Biology